Background

Asthma exacerbations in children pose a significant burden on healthcare systems and families. While traditional risk assessment tools exist, artificial intelligence (AI) offers the potential for enhanced prediction models.

Tips for taking part in sports and exercising when you have asthma.

Tenants say they have experienced mould, mushrooms growing through carpets and water leaks.

In the context of asthma severity prediction, this study delves into the feature importance of various symptoms and demographic attributes. Leveraging a comprehensive dataset encompassing symptom occurrences across varying severity levels, this investigation employs visualisation techniques, such as stacked bar plots, to illustrate the distribution of symptomatology within different severity categories. Additionally, correlation coefficient analysis is applied to quantify the relationships between individual attributes and severity levels. Moreover, the study harnesses the power of random forest and the Gini importance methodology, essential tools in feature importance analytics, to discern the most influential predictors in asthma severity prediction. The experimental results bring to light compelling associations between certain symptoms, notably 'runny-nose' and 'nasal-congestion', and specific severity levels, elucidating their potential significance as pivotal predictive indicators. Conversely, demographic factors, encompassing age groups and gender, exhibit comparatively weaker correlations with symptomatology. These findings underscore the pivotal role of individual symptoms in characterising asthma severity, reinforcing the potential for feature importance analysis to enhance predictive models in the realm of asthma management and bioinformatics.

Sacramento, CA – Almost 1 million people in California have work-related asthma, according to a new surveillance report from the California Department of Public Health.

Atlanta – As many as 2.7 million U.S. workers may have asthma caused or aggravated by workplace conditions, according to a report from the Centers for Disease Control and Prevention.

A recent report from the Centers for Disease Control and Prevention concluded that as many as 2.7 million U.S. workers may have asthma caused or aggravated by workplace conditions. And federal statistics show that about 11 million workers are exposed to at least one of several agents associated with occupational asthma.

Washington – Adults with occupational asthma face a higher risk of developing pneumococcal disease, but only 54 percent of them are vaccinated to help ward off an infection, according to a new study from NIOSH.

Atlanta — Occupational exposures may have contributed to 11 percent to 21 percent of all asthma-related deaths among 15- to 64-year-olds between 1999 and 2016, according to a recently released report from the Centers for Disease Control and Prevention.

Washington — The health care and social assistance industry has the highest percentage of workers with asthma among major industry groups, according to a recent study from NIOSH’s Respiratory Health Division.

East Lansing, MI — Estimating that up to half of the 600,000 or so adult cases of asthma in Michigan may be caused or aggravated by on-the-job exposure, the Michigan State University College of Human Medicine has launched a campaign intended to raise awareness of work-related asthma.

Macclesfield, England — Employees with asthma miss an average of almost 10% of their work hours, and most are restricted from performing their duties because of their symptoms, both of which negatively affect their emotional well-being, according to the findings of a recent study.

Manchester, England — Shift workers, particularly those working permanent night shifts, may be at elevated risk for moderate to severe asthma, according to a study led by British researchers.

London — Workers who use cleaning and disinfecting products may be more likely to develop asthma and chronic obstructive pulmonary disease – 50% and 43%, respectively – than those who don’t, results of a recent study led by British and Italian researchers show.

Washington — Workers involved in cleanup after the 2010 Deepwater Horizon oil rig disaster were significantly more likely to have been diagnosed with asthma or experienced asthma symptoms within three years of the incident, according to a new study from the National Institute of Environmental Health Sciences.

Tumwater, WA — Exposure to cannabis dust, fume or smoke can lead to the development of work-related asthma, warns a new hazard alert from the Washington State Department of Labor & Industries.

Atlanta — Exposure control, safety training and medical monitoring are all needed to prevent occupational asthma among cannabis workers, a new study concludes.

Barcelona, Spain — Exposure to printer toner, cleaning products and mold circulated in air conditioning systems – along with poor ventilation – can trigger asthma in office workers, results of a recent study out of England suggest.

If you have allergies or asthma, clean your smartphone more often to minimize reaction triggers that may be lurking on the surface, researchers recommend in a recent study.

Occupational asthma is a respiratory disease in which on-the-job exposure to certain substances causes the airways of a person’s lungs to swell and narrow.

Occupational asthma can develop when a worker breathes in gases, chemical fumes, dust or other work-related substances. According to Mayo Clinic, it also can result from exposure to a substance a worker is sensitive to, triggering an allergic or immunological response.

More than 300. That’s how many known triggers can be found in the workplace that cause or worsen asthma, according to NIOSH. 

New Brand Reflects Growth and Dedication to Making a Lasting Impact

A team of physician-scientists calculated how much greenhouse gas is being emitted by inhalers prescribed for asthma and chronic obstructive pulmonary disease.

The post Is your asthma inhaler bad for the environment? appeared first on Scope.

The allergy and asthma drug montelukast, also known as Singulair, can cause psychiatric side effects—and researchers aren’t sure why

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Background

Severe asthma significantly impacts a minority of children with asthma, leading to frequent symptoms, hospitalisations and potential long-term health consequences. However, accurate global data on severe asthma epidemiology is lacking. This study aims to address this gap, providing data on severe asthma epidemiology, regional differences and associated comorbidities.

Single maintenance and reliever therapy (SMART) is an asthma treatment approach that utilizes combined inhaled corticosteroids and long-acting β-agonists for maintenance and quick relief therapy. Despite the evidence for its benefits in asthma treatment and its adoption into American and international asthma guidelines and recommendations, SMART remains a practice of some debate. This article reviews the available evidence for SMART and offers guidance for its integration into comprehensive asthma management. Overall, short-acting β-agonist-only asthma therapy regimens should be avoided, regardless of condition severity (SOR A Recommendation). Family medicine clinicians should start SMART for patients requiring either GINA Step 3 or 4 therapy, especially if they have signs of poor adherence (SOR B Recommendation). Finally, use budesonide-formoterol over other inhaled corticosteroid/long-acting β-agonist combinations when implementing SMART (SOR B Recommendation).

COPD is a heterogeneous condition, with tobacco smoking being the main environmental risk factor. The presence of type 2 (T2) inflammation is a well-recognised feature of asthma; however, it is now apparent that a subset of COPD patients also displays evidence of T2 inflammation with respect to elevated eosinophil counts and altered gene and protein expression of several T2 inflammatory mediators. T2 inflammatory mediators represent an attractive therapeutic target in both COPD and asthma; however, the efficacy of pharmaceutical interventions varies between diseases. Furthermore, the nature of some shared clinical features also differs. We provide a narrative review of differences in the nature of T2 inflammation between COPD and asthma, which may partly explain phenotypic differences between diseases. We focus on evidence from studies of pulmonary histopathology, sputum and epithelial gene and protein expression, and response to pharmacological interventions targeted at T2 inflammation.

The UK’s first postal inhaler recycling scheme has been launched by pharmaceutical company Chiesi to support a more sustainable way of living for people with respiratory illnesses.